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    Why Women Should Lift Weights

    On a regular basis, clients tell me that they are afraid to strength train because they are afraid of getting “bulky.” When paired up with cardio and the right nutrition, strength training will NOT make you bulky.
    Fit, firm and tone is not bulky. In fact, you will more than likely drop a few sizes as your body composition changes. Where’s the bulk? It’s definitely not in the slimmer waist, tighter butt and things or those toned arms.

     
    Here are six fantastic benefits of strength training:
    1. Strength training will make you stronger. And when is strength ever a bad thing? Whether it benefits your girlfriend when you help her move, or impresses the hot guy at work when you beat him in arm wrestling, there just isn’t a downside to being strong.
    2. Building muscle gives you shape. The best part is you have the ability to put that shape EXACTLY where you want it! You can make yourself more symmetrical (which is a sign of beauty across all cultures), balance your upper body with your lower body and create the illusion of a smaller waist if you don’t already have one. Building a beautiful physique is a choice made in the gym.
    3. Strength training helps support the spine. If you find yourself with aches and pains in new places, you may lack core strength and postural support. Strengthening those muscles can alleviate those aches and pains.
    4. Standing tall with better posture can help you immediately look leaner. If you got it, you should definitely flaunt it. Chest up, boobs out, stand tall. Building overall muscle will help you stand up straighter and look more confident.
    5. Strength training makes you a badass! Confidence is sexy. And walking over to the weight rack and grabbing 25-pound dumbbells is impressive. The best part? You don’t care who is watching because you have officially left every guy in the free-weight section speechless.
    6. Muscle helps burn fat and raises your metabolism. Your body burns more calories, maintaining muscle even at rest. While this is not permission to eat whatever you want, it does allow you to control your physique.
     
    There is no downside to strength training as a girl. As for the “bulky” myth … we do not produce enough testosterone nor do we eat enough food to get bulky with muscle. Like I tell my clients: “Strength training doesn’t make you bulky, cupcakes make you bulky … ”
    Girls, if you want to feel energized, confident, stronger and more alive … strength training is for you. What about fitting into your favorite outfit from last year or the year before? Strength training. Want to tighten up that butt, slim your waistline or get back those sexy, shapely legs? You guessed it, strength training.
    Bottom line, strength training, along with good nutritional habits and cardio, should be a part of every girl’s workout! More

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    Hormones and Weight Loss

    Ladies— have you ever felt that your hormones were working against you when you were dieting and trying to lose weight? Well, perhaps you were not entirely incorrect! Oftentimes, low-calorie diets can adversely affect certain hormone levels, especially those hormones that stimulate appetite and cause increased hunger. This is frequently the reason many women wind up gaining weight back after a diet ceases. Two such hormones that do those things are called “the hunger hormones,” ghrelin and leptin.

    Ghrelin and Fat Storage
    Ghrelin, a 28-amino-acid peptide hormone that is secreted primarily by the stomach, is implicated in signaling hunger to the brain and increasing food intake. It may also stimulate fat storage. Ghrelin levels have been found to increase when a person consumes a low-calorie diet (eating less than their resting metabolic rate), and decrease when a person eats too many calories. Research is showing that the ghrelin receptors (GSR1a) and the dopamine receptors (DRD2) in the brain can interact with one another and cause a person to increase their caloric consumption, which could lead to weight gain and obesity. Ghrelin increases food intake by modulating the activity of dopaminergic neurons, and restricting food intake can increase the activity of ghrelin, increasing the number of DRD2 receptors. Studies are now finding decreased levels of dopamine DRD2 receptors in obese people compared with normal-weight people.
    The ghrelin hormone not only stimulates the brain to give rise to an increase in appetite, it also favors the accumulation of lipids in visceral fatty tissue, located in the abdominal region and considered to be the most dangerous type of adipose tissue. Excess abdominal fat is strongly associated with an increased risk for many different diseases, such as hypertension, metabolic syndrome, high cholesterol, coronary artery disease and premature death. Abdominal obesity also places people at a higher risk for developing a condition called insulin resistance, which increases the risk of type 2 diabetes.
    Leptin’s Effects on Bodyweight
    The other hormone associated with appetite is leptin. Leptin is known as the appetite suppressor and appears to have great effect on bodyweight and metabolism.
    Leptin helps signal the brain that the body has enough energy stores such as body fat. Many people who are overweight or obese have an impaired response to leptin’s signals, even though they have higher levels of the hormone in their blood, suggesting that obese individuals may be resistant to the effects of leptin. Leptin is synthesized and secreted primarily by adipocytes. It is present in blood serum in direct proportion to the amount of adipose tissue. As the fat cells become enlarged in obese individuals, they secrete a greater amount of leptin. Leptin communicates with the central nervous system to regulate energy intake and energy stores in the body so that the hypothalamus can efficiently maintain a stable bodyweight.
    Leptin Resistance
    Leptin resistance describes the apparent inconsistency of leptin’s action as an appetite-suppressing hormone and the reason for elevated levels in the majority of obese individuals. Theoretically, the human body is designed to respond to low levels of leptin, which occurs when fat stores are depleted, and not when the hormone is circulating at normal or elevated levels. There are a couple of suggested molecular mechanisms used to explain the reason for leptin resistance, such as impaired leptin transport across the blood-brain barrier or impairment of leptin receptor function and signaling. Also, it may be leptin itself playing a role in the development of resistance to its own effects, so called “leptin-induced leptin resistance.”
    Chronically raised leptin levels, which characterize obesity, decrease the transport of leptin into the central nervous system and impair the signaling properties of leptin receptors. The resulting resistance to leptin is the cause of the increased susceptibility to diet-induced obesity, which in turn raises leptin levels further and worsens existing leptin resistance leading to a vicious cycle of weight gain.
    Several factors influence the rate of leptin transport into the central nervous system. One influential factor may be α-adrenergic stimulation increasing transporter activity and decreasing transporter activity could be due to hypertriglyceridemia. Hypertriglyceridemia is generally observed during prolonged, very low calorie dieting (starvation dieting), and it is theorized that the ability of triglycerides to inhibit leptin transport may be due to their ability to counteract the stimulation of appetite suppressing signals. Conversely, hypertriglyceridemia is also associated with obesity and may in part be responsible for the impairment of leptin transport that is observed in obese individuals and defines the condition of peripheral leptin resistance.
    Cytokines
    Adipose tissue not only secretes leptin, it also secretes cytokines. Cytokines are elements of the immune system that are critical for maintaining proper levels of the immune system and they respond to inflammation in the body. Research is showing that lower levels of cytokines could also help to prevent obesity. Improvements in insulin sensitivity with weight loss are mediated by changes in inflammation in obese individuals. Improvements in glucose metabolism with weight-loss programs are independently associated with decreases in cytokine concentrations, suggesting that a reduction in inflammation is a potential mechanism that mediates improvements in insulin sensitivity.
    Cytokines respond to inflammation, and inflammation is produced by the eicosanoids, which originate from arachidonic acid. Arachidonic acid is an omega-6 fatty acid that is primarily controlled by diet. Following an anti-inflammatory diet (foods low in omega-6) may help to decrease levels of cytokines and aid in weight loss.
    Peptide YY
    Peptide YY is another protein hormone released by cells in the ileum and colon of the intestines in response to feeding that reduces appetite. Peptide YY is released in the blood and works by binding to receptors in the brain. These receptors then cause a decreased appetite and make people feel full after eating. Peptide YY also acts in the stomach and intestines to slow down the movement of food through the digestive tract. Peptide YY secretion is mainly stimulated by the presence of food in the digestive tract, particularly fat and protein. The amount of peptide YY that is released into the blood depends on the amount of calories eaten. The highest levels of peptide YY are found in the second hour after eating. Peptide YY levels then gradually decrease. Low levels of peptide YY are seen during long periods without eating, for example overnight.
    High peptide YY concentrations are unusual. They will cause a decrease in appetite and food intake. High peptide YY concentrations are associated with diseases where there is dramatic weight loss, such as anorexia nervosa, celiac disease, inflammatory bowel disease (Crohn’s disease and ulcerative colitis) and some cancers. Low peptide YY concentrations are associated with an increase in appetite and food intake. Low peptide YY levels are seen in relation to obesity and before the onset of type 2 diabetes and may contribute to weight gain in these conditions. It has also been known that obese people secrete less PYY than non-obese people. However, low peptide YY concentrations are very unlikely to be the main cause of obesity, as the levels decrease after weight gain has started. Researchers are showing that increasing the protein content of a person’s diet increases the body’s own peptide YY, helping to reduce hunger and aid in weight loss.
    Cortisol
    The hormone cortisol is produced in the adrenal cortex in response to adrenal cortical stimulating hormone (ACTH) produced in the pituitary gland. Cortisol plays an important role in regulating blood sugar, energy production, inflammation, the immune system and healing. Low cortisol levels may cause chronic fatigue, exhaustion and a disease of the endocrine system called Addison’s disease. If your adrenal glands are producing too much cortisol, you may develop conditions such as weight gain, especially around the abdomen, depressed immune function with all of the consequences, accelerated aging and stomach ulcers. Recently, a lot of attention has been directed to the effects of excess cortisol on weight gain and on the difficulty in losing weight.
    Cortisol is released in response to stress. The stress can be physical, environmental, chemical or idiopathic. All forms of stress produce the same physiological consequences. This includes environmental stress (heat, cold and noise, etc.), chemical stress (pollution, drugs, etc.), physical stress (overexertion, trauma, infection, etc.), psychological stress (worry, fear, etc.) and biochemical stress (nutritional deficiencies, refined sugar consumption, etc.). Whenever a person is subjected to a prolonged amount of stress, they are also subject to prolonged elevated levels of cortisol, which can cause the increased central adiposity and weight gain. Ways to lower cortisol levels include stress reduction, rest and eating a low glycemic diet. High dietary sugar can increase cortisol levels.
    Human Growth Hormone
    Human growth hormone (HGH) is produced by the pituitary gland and is necessary for organ and bone growth in youths. In adulthood, it is still vital for other functions. Deficiencies in HGH can cause cardiovascular disease, weight gain, decreased muscle mass, wrinkling of the skin, decreased energy and other conditions associated with the aging process. The hormone naturally decreases rapidly after age 30, and by the time a woman reaches her 40s the results are quite noticeable.
    Overall, obesity and, more importantly, fat stores in the abdominal region are indicative of impaired growth hormone production, but weight loss can help to offset this problem. High-glycemic-load carbohydrates inhibit growth hormone secretion and should be avoided. Since the majority of growth hormone secretion occurs at night during deep sleep, it is important to get a full seven to nine hours of good sound sleep. Also, your last meal of the day should contain a high-protein source and possibly a low-glycemic carbohydrate to help growth hormone secretion. Maintaining optimal levels of HGH through healthy lifestyle practices, nutrition and exercise can significantly help to increase lipolysis due to the fat-burning effects of HGH.
    Estrogen
    Estrogen actually refers to a collection of hormones that affect sexual function and reproduction, metabolism, mood, development growth and tissue health and development. There is an inverse relationship between estrogen and bodyweight during menopause, when estrogen levels drop, but weight tends to rise. Changes in estrogen receptors in the hypothalamus of the brain, a sign of menopause, means it can be harder to lose weight. In turn there is a greater burden on women to eat a healthy diet and exercise more to help their metabolism make up for the loss of estrogen, which helps regulate weight gain and appetite. Interestingly, fat cells produce estrogen, along with other hormones and proteins. But estrogen produced in fat cells will not help regulate obesity and metabolism. Instead, estrogen that accompanies weight gain, especially in abdominal fat, puts women at a higher risk for breast cancer, because estrogen stimulates cell growth.
    There is another condition associated with estrogen know as “estrogen dominance.” Estrogen dominance occurs when the body has too much estrogen, from overproduction or lack of progesterone. Estrogen dominance can result from excess estrogen production within the body, hormone replacement therapy common in menopause, and a drop in progesterone production or intake of excess estrogen from environmental sources, such as meat injected with hormones or man-made chemicals the body confuses with estrogen. Weight gain is a direct effect of estrogen dominance, especially increased fat accumulation in the abdominal region. This condition, if left unmanaged, can result in a continuous cycle of excess fat tissue producing more estrogen and estrogen producing more fatty tissue.
    The type of treatment you may need to follow to balance this hormone level depends on whether you have high or low levels. Some women entering menopause may need to have a discussion with their physician about the possibility of hormone replacement treatment, but there are many natural supplements that can aid in balancing levels of estrogen. Another way is through exercise and diet. It is no secret that exercise can help lower fat levels in your body, and since fall cells produce estrogen, exercise is a great way to possibly lower levels of the hormone. The reduction of estrogen in postmenopausal women causes an accumulation of fat, which can be counteracted by exercise as well. Many foods contain certain levels of estrogen. Food containing higher levels of estrogen (cucumbers and brown rice) can help to raise levels, whereas low estrogen containing foods (white rice and broccoli) can help to lower them.
    The Bottom Line
    There is increasing evidence showing that once people lose weight, their natural hormones may tend to push them towards regaining weight due to people’s adaptation to low-calorie dieting and being poorly adapted to proper nutrition. Changes in the appetite hormones can persist for a prolonged period of time, sometimes up to one year after dieting. Losing weight is not always a neutral event. Sometimes it is not by accident that people who lose weight wind up gaining it back.
    Many of the hormones in your body regulate metabolism and make it challenging to lose weight if those particular hormones are not balanced within your body. People struggling with their weight-loss efforts should not be discouraged by the new research suggesting that hormones are a key component in weight loss and/or weight regain. The power of success is in your hands towards achieving a healthy bodyweight by eating a nutritious diet, exercising and possibly receiving proper medical supervision if necessary.
    References
    Ashwini, O. & Giles Y. (2010). Leptin and the Control of Body Weight: A Review of Its Diverse Central Targets, Signaling Mechanisms, and Role in the Pathogenesis of Obesity . Integrative Physiology, 18(2): 221-229.
    Banks WA. (2001). Enhanced leptin transport across the blood-brain barrier by alpha 1-adrenergic agents. Brain Res, 899:209–217.
    Banks W., et al. (2004). Triglycerides induce leptin resistance at the blood-brain barrier. Diabetes, 53:1253–1260.
    Basque Research (2009, May 20). Action Of Ghrelin Hormone Increases Appetite And Favors Accumulation Of Abdominal Fat. ScienceDaily. Retrieved March 16, 2012, from http://www.sciencedaily.com­ /releases/2009/05/090520055519.htm
    Collata, G.. (2011, October 27). Study shows why it is hard to keep weight off: Biological changes last, increasing appetites. The New York Times.
    Coppack, S. (2001). Pro-inflammatory cytokines and adipose tissue. Proceedings of the Nutrition Society, 60(3): 349-356.
    Crujeiras, A. & Casanueva, F. (2012). Decreased ghrelin levels: the cause of obesity and weight regain. Endocrinology & Metabolism, 7(2): 127-129.
    Jakubowicz, D., Froy, O., wainstein, J. & Boaz, M. (2012). Meal timing and composition influence ghrelin levels, appetite scores and weight loss maintenance on overweight and obese adults. Steroids, 77(4): 323-331.
    Kern, A., Zeckler, R., Walsh, H. & Smith, R. (2012). Apo-ghrelinreceptor forms heteromers with DRD2 in hypothalamic neurons and is essential for anorexigenic effects of DRD2 agonism. Neuron, 73(2): 317-332.
    Morinigo, R., Vidal J., Lacy, A., Degado, S., Casamitijana, R., & Gomis, R. (2008). Circulating peptide YY, weight loss, and glucose homeostasis after gastric bypass surgery in morbidly obese subjects. Annals of Surgery, 247(2):270-275.
    McMurray, R. & Hackney, A. (2005). Interactions of metabolic hormones, adipose tissue and exercise. Sports Medicine, 35(5): 393-412.
    Ryan, A. & Nicklas, B. (2004). Reductions in plasma cytokine levels with weight loss improve insulin sensitivity in overweight and obese postmenopausal women. Diabetes Care, 27(7):1699-1705.
    Seim, I., El-Salhy, M., Hausken, T., Gundersen, D. & Chopin, L. (2012). Ghrelin and the brain –gut axis as a pharmacological target for appetite control. Current Pharmaceutical Design, 18(6):768-775.
    Sumithran, P., et al. (2011). Long-term persistence of hormonal adaptations to weight loss. The New England Journal of Medicine,
    Yukawa, M., et al. (2006). Effect of aging on the response of ghrelin to acute weight loss. Journal of the American Geriatrics Society, 54: 648-653. More

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    Change Your Weight Set Point

    Ever wonder why no matter how many times you diet or how much exercise you perform, your body weight somehow manages to creep back up to where it was before you started! Your weight ‘set point’ could be to blame. Research suggests that, in some circumstances, no matter how much dieting or exercising you do, the body will eventually […] More

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    6 Reasons Women Should Lift Weights

    On a regular basis, clients tell me that they are afraid to strength train because they are afraid of getting “bulky.” When paired up with cardio and the right nutrition, strength training will NOT make you bulky. Fit, firm and tone is not bulky. In fact, you will more than likely drop a few sizes as your […] More